New PDF release: Animal Models in Psychiatry, II

By Hymie Anisman, Steve Zalcman, Nola Shanks (auth.), Alan A. Boulton, Glen B. Baker, Mathew T. Martin-Iverson (eds.)

ISBN-10: 0896031772

ISBN-13: 9780896031777

ISBN-10: 159259624X

ISBN-13: 9781592596249

The Animal types in Psychiatry volumes are loosely prepared by way of topic. the 1st quantity encompasses a variety of chapters fascinated with schizophrenia, psyc- ses, neuroleptic-induced tardive dyskinesias, and different d- orders that can contain dopamine, equivalent to cognizance deficit sickness and mania. additionally integrated is a bankruptcy describing a behavioral version for activity-induced anorexia. the second one quantity offers with affective and anxiousness issues, but additionally comprises chapters on matters now not simply labeled as both psychotic, affective, or anxiety-related, similar to aggression, psychological retardation, and reminiscence problems. 4 chapters on animal types of schizophrenia or psychoses are integrated in quantity 18 end result of the value of those issues in psychiatry. Likewise, 3 chapters within the current v- ume take care of affective issues, with a fourth bankruptcy on circadian rhythms that still contributes to equipment for a- mal versions in affective issues. Following the 1st 4 chapters are chapters facing types of hysteria and panic, chapters on aggression, one on psychological retardation, and a last bankruptcy overlaying reminiscence problems. a number of the behaviorally-based versions of affective problems contain inducing rigidity in a- mals, often on a prolonged foundation. the 1st bankruptcy by way of Anisman, Zalcman, Shanks, and Zacharko describes a few of the neurochemical results which are linked to the power program of sensors.

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Animals, thus leading to an apparent enhancement of the immune response. It will be recognized, however, that the immunopotentiation may, in fact, be disadvantageous in that it may lend itself to enhanced autoantibody activity (seeFig. 3). A series of experiments conducted by Ader and his associates (seeAder and Cohen, 1985) suggested that immune functioning may be subject to conditioning. In particular, it was demonstrated that exposure to saccharin some time after pairing of this novel taste with the immunosuppressant, cyclophosphamide, resulted in a reduction of the PFC response (Ader and Cohen, 1985).

Specifically, the characteristics of the shock and swim situations, as well as the response styles that could be adopted in the two tasks were markedly different from one another. It is our contention that the various behavioral consequences associated with stressors may be subserved by different neurochemical mechanisms and subject to differential rates of adaptation. As will be seen later, Animal Model of Depression 37 several of the behavioral consequences of stressors are independent of one another, and manipulations that affect one behavior may be ineffective in altering other behaviors.

Thus, the prevention of the DA reductions following a chronic stressor may be attributable to moderation of the excessive release of DA that is ordinarily engendered by an acute stressor. 3. , 1968a; seealso Wilkinson and Jacobs, 1988). , 1983). , 1975). More recently, it was reported that a 15min session of footshockincreased accumulation of 5-HIAA and reduced concentrations of 5-I-U in the mesocortex and in the hypothalamus of CD-l mice. When a more protracted stressor period was employed (30 min), amine utilization increased still further, as reflected by greater accumulation of 5-HIAA; however, 5-HT levels were comparable to those of nonstressed animals, whereas brain tryptophan levels increased appreciably 14 Anisman et al.

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Animal Models in Psychiatry, II by Hymie Anisman, Steve Zalcman, Nola Shanks (auth.), Alan A. Boulton, Glen B. Baker, Mathew T. Martin-Iverson (eds.)


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